eMedicine Specialties > Sports Medicine > Knee

Posterior Cruciate Ligament Injury

Charles S Peterson, MD, Consulting Staff, Arizona Sports Medicine Center
Thomas Agesen, MD, Assistant Clinical Professor, UMDNJ, New Jersey Medical School; Consulting Staff, Department of Physical Medicine and Rehabilitation, Mountainside Hospital, Summit Overlook Hospital; Janos P Ertl, MD, Clinical Assistant Professor, Department of Orthopedic Surgery, Chief of Orthopedic Trauma, University of California at Davis; Director of Amputee Clinic, Kaiser Hospital; Gyorgy Kovacs, MD, Department of Orthopedic Surgery, Consulting Surgeon, GOC Clinic
Contributor Information and Disclosures

Updated: Dec 22, 2006

Introduction

Background

The posterior cruciate ligament (PCL) is described as the primary stabilizer of the knee by many authors. PCL injuries are less common than anterior cruciate ligament (ACL) injuries, and they often go unrecognized. The PCL is broader and stronger than the ACL and has a tensile strength of 2000 N. Injury most often occurs when a force is applied to the anterior aspect of the proximal tibia when the knee is flexed. Hyperextension and rotational or varus/valgus stress mechanisms also may be responsible for PCL tears. Injuries may be isolated or combined with other ligamentous injuries. A PCL tear can result in varying degrees of disability, from no impairment to severe impairment.  PCL injury has been overly simplified, and the functional disability of PCL injury may be underestimated (Wang, 2002). 

The primary function of the PCL is to prevent posterior translation of the tibia on the femur. The PCL also plays a role as a central axis controlling and imparting rotational stability to the knee. This injury has received little attention in the past, compared with the ACL; however, this emphasis on the ACL has stimulated increased interest in the treatment of PCL injuries. Controversy regarding treatment of isolated PCL injuries exists in the literature, with recommendations supporting both operative and nonoperative therapy. Current management of PCL injuries unfortunately can yield relatively poor clinical outcomes, whether surgically or conservatively treated (Margheritini, 2002).

Frequency

United States

True incidence in the United States is unknown. In National Football League predraft physical examinations, a 2% incidence of isolated, asymptomatic, and unknown PCL injuries was found; operated, isolated, and combined PCL injuries were reported at an incidence of 3.5-20%. On the KT-1000 stress test examination, a 7% incidence of PCL injuries was found, of which 40% were isolated and unidirectional and 60% were multidirectional.

Functional Anatomy

The PCL originates from the intercondylar notch of the femur on the roof of the medial femoral condyle. The insertion is central on the posterior aspect of the tibial plateau, on a depression between the tibial plateaus, extending 1 cm below the articular surface (Sheps, 2005). The ligament is composed of a larger anterolateral bundle and a smaller posteromedial bundle. The anterior component is tightest in the midarc of flexion and the posterior fibers are tight in extension and deep flexion.

In addition, variable anterior and posterior meniscofemoral ligaments of Humphrey and Wrisberg attach distally and proximally to the PCL, respectively. The meniscofemoral ligaments attach distally to the posterior horn of the lateral meniscus, in a slanting orientation, providing resistance to the tibial posterior drawer (Amis, 2006). The PCL is an extrasynovial structure that lies behind the intra-articular portion of the knee. The primary function of the PCL is to resist posterior displacement of the tibia in relation to the femur; its secondary function is to prevent hyperextension and limit internal and varus/valgus rotation.

Sport Specific Biomechanics

Disruption may occur with forced hyperextension while the foot is planted in dorsiflexion. A force applied to the anteromedial aspect of the knee, as during a football tackle, results in a posteriorly directed force and a varus hyperextension force, leading to PCL and posterolateral capsular ruptures.

Clinical

History

  • Knowledge of the mechanism of injury is helpful. The following 4 mechanisms of PCL injury are recognized:
    • A posteriorly directed force on a flexed knee, eg, the anterior aspect of the flexed knee striking a dashboard, may cause PCL injury.
    • A fall onto a flexed knee with the foot in plantar flexion and the tibial tubercle striking the ground first, directing a posterior force to the proximal tibia, may result in injury to the PCL.
    • Hyperextension alone may lead to an avulsion injury of the PCL from the origin. This kind of injury may be amenable to repair.
    • An anterior force to the anterior tibia in a hyperextended knee with the foot planted results in combined injury to the knee ligaments along with knee dislocation.
  • In chronic PCL tears, discomfort may be experienced with the following positions or activities:
    • A semiflexed position, as with ascending or descending stairs or an incline
    • Starting a run
    • Lifting a load
    • Walking longer distances
  • Retropatellar pain symptoms may be reported as a result of posterior tibial sagging.
  • Swelling and stiffness may be reported in cases of chondral damage.
  • Individuals may describe a sensation of instability when walking on uneven ground
  • Medial joint line pain may be reported.

Physical

In the acute stage of isolated PCL injuries, symptoms usually are vague and minimal. The following physical examination findings are common in individuals who have sustained PCL injuries:

  • Minimal to no pain
  • Minimal hemarthrosis
  • Usually full or functional range of motion (ROM)
  • Contusion over the anterior tibia
  • Posterior tibial sag
    • To observe posterior tibial sag, place patient supine and put 90° of flexion at the knee and hip. In such a position, gravity pulls posteriorly on the tibia, and in the case of PCL disruption, the tibia falls even or behind the femoral condyles. Comparison should be made to the opposite knee.
    • Grade I injury is indicated when side-to-side asymmetry exists but the tibial plateau is anterior to the femoral condyles. Grade II injury occurs when the tibial plateau is even with the femoral condyles, and grade III injury occurs when the tibial plateau falls behind the femoral condyles.
  • Posterior sag sign during extension
    • The patient is supine on the examining table, with the examiner at the end of the table. The examiner supports both of the patient's heels simultaneously with legs in full extension.
    • If a posterior sag can be seen on the injured side compared to the other side, there usually is an injury to the PCL and some secondary restraint (ie, medial collateral ligament [MCL], lateral collateral ligament [LCL], posterolateral corner).
  • Positive quadriceps active test
    • During the quadriceps active test, the patient is placed supine with the knee flexed to 90° and the foot placed flat on the examining table.
    • If an individual with an intact PCL is in such a position with the quadriceps relaxed, the tibia is 10 mm anterior to the femoral condyles. If there is a PCL disruption, gravity pulls the tibia even or behind the femoral condyles, with the quadriceps relaxed. The examiner restrains the ankle from moving, and the patient is asked to contract the quadriceps. In individuals who have a deficient PCL, the tibia moves forward; if the tibia moves forward more than 2 mm, the quadriceps active test is positive.
  • Findings of the posterior drawer test
    • The posterior drawer test is considered the most useful for documenting PCL injury.
    • The patient is placed supine with both knees flexed to 90° and the feet in neutral rotation placed flat on the table (examiner must compare side-to-side difference). As mentioned previously, in such a position the tibial plateau should be about 10 mm anterior to the femoral condyles.
    • The examiner imparts a posterior force to the proximal tibia, and if the tibia can be displaced 0-5 mm or if there is side-to-side asymmetry, a grade I injury is indicated. If the tibia can be displaced 5-10 mm or the tibial plateau can move posteriorly even with femoral condyles, a grade II injury is indicated. If the tibia can be moved more than 10 mm posteriorly or the tibial plateau moves behind the femoral condyles, a grade III injury is indicated.
    • The internal and external rotation of the foot during the posterior drawer test can assess different structures. If the foot is placed in internal rotation, the PCL and tibial collateral ligaments are tested. If the foot is placed in external rotation, the PCL, LCL, and posterolateral corner are tested. Assessment of the posterolateral corner is paramount with PCL injuries because isolated PCL injuries have a very good prognosis. However, a PCL injury combined with posterolateral corner injury has a less favorable prognosis. The external rotation recurvatum test and the reverse pivot shift test (described below) are used to assess the posterolateral corner.
  • Findings of the external rotation recurvatum test: This test is the same as the posterior sag sign described above, except the examiner notices significant subluxation of the lateral tibial plateau.
  • Findings of the posterolateral drawer test in 90° of flexion: This test is performed with the patient sitting with thighs supported by the examining table and legs lying off the end of the examining table. In such a position, the knees are at 90° of flexion. The examiner performs a posterior drawer test. If the posterolateral structures are injured, the lateral tibial plateau rotates posteriorly around the axis of the PCL as the posterior force is applied.
  • False-positive Lachman test: The Lachman test is performed to assess the integrity of the ACL. In a knee with a deficient PCL, the starting position of the tibial plateau is posterior to normal. Since the starting point is posterior, there seems to be increased anterior laxity. This results in a false-positive Lachman test. The endpoint of the Lachman test is still firm with PCL disruption.

Causes

Possible causes of PCL injuries include the following:

  • Football injuries
  • Running injuries
  • Motor vehicle accidents
  • Falls onto a flexed knee

Contents

Overview: Posterior Cruciate Ligament Injury
Differential Diagnoses & Workup: Posterior Cruciate Ligament Injury
Treatment & Medication: Posterior Cruciate Ligament Injury
Follow-up: Posterior Cruciate Ligament Injury
Multimedia: Posterior Cruciate Ligament Injury

References

  1. Ahn JH, Nha KW, Kim YC, Lim HC, Nam HW, Wang JH. Arthroscopic femoral tensioning and posterior cruciate ligament reconstruction in chronic posterior cruciate ligament injury. Arthroscopy. Mar 2006;22(3):341.e1-4. [Medline].

  2. Ahn JH, Yoo JC, Wang JH. Posterior cruciate ligament reconstruction: double-loop hamstring tendon autograft versus Achilles tendon allograft--clinical results of a minimum 2-year follow-up. Arthroscopy. Aug 2005;21(8):965-9. [Medline].

  3. Amis AA, Gupte CM, Bull AM, Edwards A. Anatomy of the posterior cruciate ligament and the meniscofemoral ligaments. Knee Surg Sports Traumatol Arthrosc. Mar 2006;14(3):257-63. [Medline].

  4. Baker CL Jr, Norwood LA, Hughston JC. Acute combined posterior cruciate and posterolateral instability of the knee. Am J Sports Med. May-Jun 1984;12(3):204-8. [Medline].

  5. Bergfeld JA, McAllister DR, Parker RD, et al. The effects of tibial rotation on posterior translation in knees in which the posterior cruciate ligament has been cut. J Bone Joint Surg Am. Sep 2001;83-A(9):1339-43. [Medline].

  6. Browner BD. Skeletal Trauma: Fractures, Dislocations, Ligamentous Injuries. Philadelphia, Pa: WB Saunders Co; 1998.

  7. Cain TE, Schwab GH. Performance of an athlete with straight posterior knee instability. Am J Sports Med. Jul-Aug 1981;9(4):203-8. [Medline].

  8. Clancy WG Jr, Shelbourne KD, Zoellner GB, et al. Treatment of knee joint instability secondary to rupture of the posterior cruciate ligament. Report of a new procedure. J Bone Joint Surg Am. Mar 1983;65(3):310-22. [Medline].

  9. Cosgarea AJ, Jay PR. Posterior cruciate ligament injuries: evaluation and management. J Am Acad Orthop Surg. Sep-Oct 2001;9(5):297-307. [Medline].

  10. Cross MJ, Powell JF. Long-term followup of posterior cruciate ligament rupture: a study of 116 cases. Am J Sports Med. Jul-Aug 1984;12(4):292-7. [Medline].

  11. Daniel DM, Stone ML, Barnett P, Sachs R. Use of the quadriceps active test to diagnose posterior cruciate-ligament disruption and measure posterior laxity of the knee. J Bone Joint Surg Am. Mar 1988;70(3):386-91. [Medline].

  12. Delee JC. Orthopaedic Sports Medicine, Principles and Practice. Vol 2. Philadelphia, Pa: WB Saunders Co; 1994.

  13. Donovan. Posterior cruciate ligament injury on artificial turf. Orthop. 1977;1:20.

  14. Fowler PJ, Messieh SS. Isolated posterior cruciate ligament injuries in athletes. Am J Sports Med. Nov-Dec 1987;15(6):553-7. [Medline].

  15. Harner, Miller. Complex topics in knee surgery. Clin Sports Med. 1999;18(1).

  16. Janousek AT, Jones DG, Clatworthy M, et al. Posterior cruciate ligament injuries of the knee joint. Sports Med. Dec 1999;28(6):429-41. [Medline].

  17. Jung TM, Höher J, Weiler A. Screw fixation of a 4 1/2-year-old PCL avulsion injury. Knee Surg Sports Traumatol Arthrosc. May 2006;14(5):469-72. [Medline].

  18. Jung YB, Jung HJ, Tae SK, Lee YS, Yang DL. Tensioning of remnant posterior cruciate ligament and reconstruction of anterolateral bundle in chronic posterior cruciate ligament injury. Arthroscopy. Mar 2006;22(3):329-38. [Medline].

  19. Lenschow S, Zantop T, Weimann A, Lemburg T, Raschke M, Strobel M. Joint kinematics and in situ forces after single bundle PCL reconstruction: a graft placed at the center of the femoral attachment does not restore normal posterior laxity. Arch Orthop Trauma Surg. May 2006;126(4):253-9. [Medline].

  20. Margheritini F, Rihn J, Musahl V, Mariani PP, Harner C. Posterior cruciate ligament injuries in the athlete: an anatomical, biomechanical and clinical review. Sports Med. 2002;32(6):393-408. [Medline].

  21. Ogata K, McCarthy JA, Dunlap J, Manske PR. Pathomechanics of posterior sag of the tibia in posterior cruciate deficient knees. An experimental study. Am J Sports Med. Nov-Dec 1988;16(6):630-6. [Medline].

  22. Paddu G, Gianni E, Chambat P. The axial view in evaluating tibial translation in cases of insufficiency of the posterior cruciate ligament. Arthroscopy. 2000;16(2):217-220.

  23. Parolie JM, Bergfeld JA. Long-term results of nonoperative treatment of isolated posterior cruciate ligament injuries in the athlete. Am J Sports Med. Jan-Feb 1986;14(1):35-8. [Medline].

  24. Safran MR, Allen AA, Lephart SM, et al. Proprioception in the posterior cruciate ligament deficient knee. Knee Surg Sports Traumatol Arthrosc. 1999;7(5):310-7. [Medline].

  25. Sekiya JK, West RV, Ong BC, Irrgang JJ, Fu FH, Harner CD. Clinical outcomes after isolated arthroscopic single-bundle posterior cruciate ligament reconstruction. Arthroscopy. Sep 2005;21(9):1042-50. [Medline].

  26. Servant CT, Ramos JP, Thomas NP. The accuracy of magnetic resonance imaging in diagnosing chronic posterior cruciate ligament injury. Knee. Aug 2004;11(4):265-70. [Medline].

  27. Shelbourne KD, Davis TJ, Patel DV. The natural history of acute, isolated, nonoperatively treated posterior cruciate ligament injuries. A prospective study. Am J Sports Med. May-Jun 1999;27(3):276-83. [Medline].

  28. Shelbourne KD, Jennings RW, Vahey TN. Magnetic resonance imaging of posterior cruciate ligament injuries: assessment of healing. Am J Knee Surg. 1999;12(4):209-13. [Medline].

  29. Sheps DM, Otto D, Fernhout M. The anatomic characteristics of the tibial insertion of the posterior cruciate ligament. Arthroscopy. Jul 2005;21(7):820-5. [Medline].

  30. Siliski JM. Traumatic Disorders of the Knee. New York, NY: Springer-Verlag; 1994.

  31. St Pierre P, Miller MD. Posterior cruciate ligament injuries. Clin Sports Med. Jan 1999;18(1):199-221, vii. [Medline].

  32. Strobel MJ, Weiler A, Schulz MS, Russe K, Eichhorn HJ. Arthroscopic evaluation of articular cartilage lesions in posterior-cruciate-ligament-deficient knees. Arthroscopy. Mar 2003;19(3):262-8. [Medline].

  33. Veltri DM, Warren RF. Isolated and combined posterior cruciate ligament injuries. J Am Acad Orthop Surg. Nov 1993;1(2):67-75.

  34. Wang CJ. Injuries to the posterior cruciate ligament and posterolateral instabilities of the knee. Chang Gung Med J. May 2002;25(5):288-97. [Medline].

Further Reading

Keywords

posterior cruciate ligament, PCL injury, PCL tear, posterior knee instability, posterior laxity of the knee

Contributor Information and Disclosures

Author

Charles S Peterson, MD, Consulting Staff, Arizona Sports Medicine Center
Charles S Peterson, MD is a member of the following medical societies: American Academy of Family Physicians, American College of Sports Medicine, and American Medical Society for Sports Medicine
Disclosure: Nothing to disclose

Coauthor

Thomas Agesen, MD, Assistant Clinical Professor, UMDNJ, New Jersey Medical School; Consulting Staff, Department of Physical Medicine and Rehabilitation, Mountainside Hospital, Summit Overlook Hospital
Thomas Agesen, MD is a member of the following medical societies: American Academy of Physical Medicine and Rehabilitation, American College of Sports Medicine, and Physiatric Association for Spine, Sports and Occupational Rehabilitation
Disclosure: Nothing to disclose

Janos P Ertl, MD, Clinical Assistant Professor, Department of Orthopedic Surgery, Chief of Orthopedic Trauma, University of California at Davis; Director of Amputee Clinic, Kaiser Hospital
Janos P Ertl, MD is a member of the following medical societies: American Academy of Orthopaedic Surgeons, Hungarian Medical Association of America, Orthopaedic Trauma Association, and Sierra Sacramento Valley Medical Society
Disclosure: Nothing to disclose

Gyorgy Kovacs, MD, Department of Orthopedic Surgery, Consulting Surgeon, GOC Clinic
Disclosure: Nothing to disclose

Medical Editor

Gerard A Malanga, MD, Associate Professor, Department of Physical Medicine and Rehabilitation, New Jersey Medical School; Director of Pain Management, University of Medicine and Dentistry at New Jersey, Overlook Hospital; Director of Sports Medicine, Mountainside Hospital
Gerard A Malanga, MD is a member of the following medical societies: American Academy of Pain Medicine, American Academy of Physical Medicine and Rehabilitation, American College of Sports Medicine, North American Spine Society, and Physiatric Association for Spine, Sports and Occupational Rehabilitation
Disclosure: Nothing to disclose

Pharmacy Editor

Francisco Talavera, PharmD, PhD, Senior Pharmacy Editor, eMedicine
Disclosure: Nothing to disclose

Managing Editor

Russell D White, MD, Professor of Medicine, Department of Community and Family Medicine, University of Missouri-Kansas City School of Medicine, Truman Medical Center Lakewood
Disclosure: Nothing to disclose

CME Editor

Jon B Whitehurst, MD, Clinical Instructor of Surgery, University of Illinois College of Medicine; Consulting Staff, Rockford Orthopedic Associates
Jon B Whitehurst, MD is a member of the following medical societies: American Academy of Orthopaedic Surgeons, American Orthopaedic Society for Sports Medicine, and Arthroscopy Association of North America
Disclosure: Nothing to disclose

Chief Editor

Craig C Young, MD, Medical Director of Sports Medicine, Sports Medicine Fellowship Director, Professor, Departments of Orthopedic Surgery and Community and Family Medicine, Medical College of Wisconsin
Craig C Young is a member of the following medical societies: American Academy of Family Physicians, American College of Sports Medicine, American Medical Society for Sports Medicine, Phi Beta Kappa, and Wilderness Medical Society
Disclosure: Nothing to disclose

 
 
HONcode

We subscribe to the
HONcode principles of the
Health On the Net Foundation

All material on this website is protected by copyright, Copyright© 1994- by Medscape.
This website also contains material copyrighted by 3rd parties.

DISCLAIMER: The content of this Website is not influenced by sponsors. The site is designed primarily for use by qualified physicians and other medical professionals. The information contained herein should NOT be used as a substitute for the advice of an appropriately qualified and licensed physician or other health care provider. The information provided here is for educational and informational purposes only. In no way should it be considered as offering medical advice. Please check with a physician if you suspect you are ill.